Prolonged Sleep Fragmentation of Mice Exacerbates Febrile Responses to Lipopolysaccharide

This study shows that prolonged sleep disruption of mice exacerbates febrile responses to lipopolysaccharide. This device provides a method to determine mechanisms by which chronic insufficient sleep contributes to the etiology of many pathologies, particularly those with an inflammatory component.

http://www.ncbi.nlm.nih.gov/pubmed/23872243